Cholecystokinin-8 vs Ibutamoren

Head-to-head comparison of Cholecystokinin-8 (Cholecystokinin (sulfated C-terminal octapeptide, CCK-8)) and Ibutamoren (Ibutamoren mesylate (MK-677)) — benefits, dosing, side effects, research data, and where to buy.

Research Score

PropertyCholecystokinin-8Ibutamoren
CategoryEndocrine PeptidesGrowth Hormone
Full NameCholecystokinin (sulfated C-terminal octapeptide, CCK-8)Ibutamoren mesylate (MK-677)
Molecular Weight1143.2 DaN/A
Half-Life1-2 minabout 24 hours
Amino AcidsDY(SO3H)MGWMDF-NH2N/A
Typical Dosephysiologic low-picomolar-nanomolar range|research-dependent10-25 mg/day
Routeendogenous secretion|research IV/SCOral
Purity≥98%≥98%
Studies Count97180
Research StatusEndogenousClinical

Cholecystokinin-8 Benefits

  • gallbladder contraction
  • pancreatic enzyme release
  • satiety signaling

Ibutamoren Benefits

  • raises GH/IGF-1
  • increases appetite
  • supports lean mass retention
  • oral non-peptide secretagogue

Cholecystokinin-8 Dosing

physiologic secretion|research-dependent

Ibutamoren Dosing

10-25 mg orally once daily|take consistently at the same time each day|monitor fasting glucose and edema in longer studies

Cholecystokinin-8 Side Effects

  • abdominal cramping
  • nausea
  • biliary contraction

Ibutamoren Side Effects

  • increased appetite
  • water retention/edema
  • somnolence or lethargy

Research Overview

Cholecystokinin-8

CCK-8 is a canonical gut hormone fragment with extensive documentation in digestive physiology and appetite control. Sulfation of the tyrosine residue is important for high-affinity receptor activity.

Ibutamoren

Clinical studies show oral ibutamoren reliably elevates GH and IGF-1 over 24 hours with once-daily dosing. Body composition and appetite effects vary by population, and development has been limited by metabolic side effects.

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Common Stacking Partners

Cholecystokinin-8 stacks with:

gallbladderpancreassatiety

Ibutamoren stacks with:

Resistance trainingHigh-protein diet
gut-hormonesatiety-peptideendocrine-peptideghrelin-receptor-agonistgh-secretagogueoral